Going to present the other side today:
This is about the mechanisms by which mood/behavioral changes in AAS users happen. It's not strictly related to testosterone/estrogen/prolactin levels and then those hormones acting on brain chemistry. The AAS themselves independent of estro/prolactin are having effects on various parts of the brain, neurotransmitters, and brain chemistry. No matter how well you manage ancillaries, you can't prevent this.
http://www.mindandmuscle.net/article...ndreas-martin/
Cliffs: "Roid rage"
can be a thing. It can also manifest with just disinhibition/increased confidence. 19-nor derived AAS are more likely to cause aggression than others, and trenbolone is more known for this than nandrolone due to the fact that, while both increase mesolimbic activity, nandrolone offsets this with estrogenic activity in the ventro medial hypothalamus.
Nandrolones
can have a positive impact on short term memory in cases where serotonin receptors have been rewired and permanently downregulated (through long term SSRI use or having too much fun raving when you were a teenager or a creepy old guy trying to get underage pussy at a rave).
There are
some potential procognitive effects for those with neuro-degenerative diseases when combined with other medicines, but this part is really speculative.
Seems like there's not a lot of great info on AAS in endocrinology journals, but there is a surprising amount in neuroscience publications. It is definitely a lot more scary playing with substances that can measurably alter the chemistry of your actual brain rather than just strictly your endocrine system. Yeah yeah yeah I know the two are linked blah blah blah. On the other hand, alcohol marijuana benadryl etc etc.
Last edited by Evoken; 06-19-2015 at 01:48 AM.